| GHS 분류 | DangerH220: Extremely flammable gas [Danger Flammable gases]P203, P210, P222, P280, P377, P381, and P403 (click each P-code to see the statement)This chemical does not meet GHS hazard criteria for 0.1% (2 of 1422) of reports.H220 (99.8%): Extremely flammable gas [Danger Flammable gases] |
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| 규제 정보 | Chemical: HydrogenRegulation (EC) No 1935/2004 (amended)Hydrogen is listed on the EPA's Chemical Data Reporting (CDR) system. Manufacturers and importers of Hydrogen are required to report information about their production and use of this chemical to the EPA under the Toxic Substances Control Act (TSCA). (40 eCFR Part 711)Hydrogen: HSNO Approval: HSR001002 Approved with controlsThe New Jersey Worker and Community Right to Know Act requires public and private employers to provide information about hazardous substances at their workplaces. (N.J.S.A. 34:5A-1 et. seq.) |
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| 기타 안전 정보 | IMAP assessments - Hydrogen: Human health tier I assessment |
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| 운송 라벨(DOT) | Flammable Gas |
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| 화재 위험 | Excerpt from ERG Guide 115 [Gases - Flammable (Including Refrigerated Liquids)]:Behavior in Fire: Burns with an almost invisible flame. (USCG, 1999)· EXTREMELY FLAMMABLE.Extremely flammable. Many reactions may cause fire or explosion. Gas/air mixtures are explosive. |
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| 화재 가능성 | Highly dangerous fire and severe explosion hazard when exposed to heat, flame, or oxidizers.... Sudden release of hydrogen into the atmosphere from storage above 79 bars may cause spontaneous ignition. ... |
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| 건강 유해성 | Excerpt from ERG Guide 115 [Gases - Flammable (Including Refrigerated Liquids)]:If atmosphere does not contain enough oxygen, inhalation can cause dizziness, unconsciousness, or even death. Contact of liquid with eyes or skin causes freezing similar to burn. (USCG, 1999)· Vapors may cause dizziness or asphyxiation without warning, especially when in closed or confined areas. |
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| 유해성 요약 | Simple asphyxiantContact with evaporating liquid can cause frostbite[HSDB] |
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| 운송 번호 및 가이드(DOT) | 1966 1151049 115 |
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| 반응성 그룹 | Reducing Agents, Weak |
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| EC 분류 | Symbol: F+R: 12S: (2)-9-16-33 |
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| UN 분류 | UN Hazard Class: 2.1 |
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| Special Reports | Harper SR, Pohland FGRecent developments in hydrogen management during anaerobic biological wastewater treatmentBiotechnol Bioeng 28 (4): 585-602 (1986). |
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| 소화 | Excerpt from ERG Guide 115 [Gases - Flammable (Including Refrigerated Liquids)]:Shut off supplyif not possible and no risk to surroundings, let the fire burn itself out. In other cases extinguish with water spray, powder, carbon dioxide. In case of fire: keep cylinder cool by spraying with water. Combat fire from a sheltered position. |
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| 응급 조치 | Fresh air, rest.ON FROSTBITE: rinse with plenty of water, do NOT remove clothes. Refer immediately for medical attention.ON FROSTBITE: rinse with plenty of water. Refer immediately for medical attention. |
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| 누출 비상 조치 | · CALL 911. Then call emergency response telephone number on shipping paper. If shipping paper not available or no answer, refer to appropriate telephone number listed on the inside back cover.· ELIMINATE all ignition sources (no smoking, flares, sparks or flames) from immediate area. |
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| 응급 처치 | Excerpt from ERG Guide 115 [Gases - Flammable (Including Refrigerated Liquids)]:The only effect of exposure to liquid hydrogen is that caused by its unusually low temperature and its action as a simple asphyxiant.General First Aid: |
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| 안전 보관 | Fireproof. Cool. Ventilation along the floor and ceiling. Separated from oxidizing materials. |
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| 노출 관리 및 개인 보호 | · Wear positive pressure self-contained breathing apparatus (SCBA). |
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| 소화 절차 | To fight fire, stop flow of gas.Approach fire with caution as high-temperature flame is practically invisible. Stop flow of gas before extinguishing fire. Use water spray to keep fire-exposed containers cool. Use flooding quantities of water as fog or spray. /Hydrogen, refrigerated liquid/Use water spray, alcohol-resistant foam, dry chemical or carbon dioxide. Wear self-contained breathing apparatus for firefighting if necessary. Use water spray to cool unopened containers. |
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| 보관 조건 | Store in a cool, dry, well-ventilated location. Outside or detached storage is preferred. Isolate from oxygen, halogens, other oxidizing materials. /Hydrogen, refrigerated liquid/A safe storage method for hydrogen for possible use as automotive fuel involves the use of metal hydrides from which the hydrogen is released at specified temperatures. Iron titanium hydride has been found the most satisfactory.Keep container tightly closed in a dry and well-ventilated place. Contents under pressure. Storage class (TRGS 510): Gases |
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| 정리 방법 | ACCIDENTAL RELEASE MEASURES: Personal precautions, protective equipment and emergency procedures: Avoid breathing vapors, mist or gas. Ensure adequate ventilation. Remove all sources of ignition. Evacuate personnel to safe areas. Beware of vapors accumulating to form explosive concentrations. Vapors can accumulate in low areas. Environmental precautions: Prevent further leakage or spillage if safe to do so. Do not let product enter drains. Methods and materials for containment and cleaning up: Clean up promptly by sweeping or vacuum.Eliminate all ignition sources. Approach release from upwind. Stop or control the leak, if this can be done without undue risk. Use water spray to disperse vapors and protect personnel. /Hydrogen, refrigerated liquid/ |
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| 비화재 누출 대응 | Excerpt from ERG Guide 115 [Gases - Flammable (Including Refrigerated Liquids)]: |
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| 처리 방법 | SRP: Recycle any unused portion of the material for its approved use or return it to the manufacturer or supplier. Ultimate disposal of the chemical must consider: the material's impact on air qualitypotential migration in air, soil or watereffects on animal, aquatic and plant lifeand conformance with environmental and public health regulations. If it is possible or reasonable use an alternative chemical product with less inherent propensity for occupational harm/injury/toxicity or environmental contamination.Product: Contact a licensed professional waste disposal service to dispose of this material. Burn in a chemical incinerator equipped with an afterburner and scrubber but exert extra care in igniting as this material is highly flammable. Offer surplus and non-recyclable solutions to a licensed disposal company. Contaminated packaging: Dispose of as unused product |
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| 누출 처리 | Evacuate danger area! Consult an expert! Ventilation. Remove all ignition sources. Remove vapour with fine water spray. |
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| 상호 작용 | Substantial evidence indicates that molecular hydrogen (H2) has beneficial vascular effects because of its antioxidant and/or anti-inflammatory effects. Thus, hydrogen-rich water may prove to be an effective anti-aging drink. This study examined the effects of H2 on endothelial senescence and clarified the mechanisms involved. Hydrogen-rich medium was produced by a high-purity hydrogen gas generator. Human umbilical vein endothelial cells (HUVECs) were incubated with 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) for various time periods in normal or hydrogen-rich medium. The baseline H2concentration in hydrogen-rich medium was 0.55 +/- 0.07 mmol/L. This concentration gradually decreased, and H2 was almost undetectable in medium after 12 hr. At 24 hr after TCDD exposure, HUVECs treated with TCDD exhibited increased 8OHdG and acetyl-p53 expression, decreased nicotinamide adenine dinucleotide (NAD(+))/NADH ratio, impaired Sirt1 activity, and enhanced senescence-associated beta-galactosidase. However, HUVECs incubated in hydrogen-rich medium did not exhibit these TCDD-induced changes accompanying Nrf2 activation, which was observed even after H2 was undetectable in the medium. Chrysin, an inhibitor of Nrf2, abolished the protective effects of H2 on HUVECs. H2 has long-lasting antioxidant and anti-aging effects on vascular endothelial cells through the Nrf2 pathway, even after transient exposure to H2. Hydrogen-rich water may thus be a functional drink that increases longevity. /Hydrogen-rich water/We observed the effect of hydrogen-rich medium on lipopolysaccharide (LPS)-induced human umbilical vein endothelial cells (HUVECs), hyaline leukocyte conglutination, and permeability of the endothelium. Endotheliocytes were inoculated on 6-well plates and randomly divided into 4 groups: control, H2, LPS, LPS+H2, H2, and LPS+H2 in saturated hydrogen-rich medium. We applied Wright's staining to observe conglutination of hyaline leukocytes and HUVECs, flow cytometry to determine the content of vascular cell adhesion protein 1 (VCAM-1) and intercellular adhesion molecule 1 (ICAM-1), enzyme-linked immunosorbent assay to measure the E-selectin concentration in the cell liquor, the transendothelial electrical resistance (TEER) to test the permeability of endothelial cells, and Western blot and immunofluorescence to test the expression and distribution of vascular endothelial (VE)-cadherin. Compared with control cells, there was an increase in endothelium-hyaline leukocyte conglutination, a reduction in VCAM-1, ICAM-1, and E-selectin, and the TEER value increased obviously. Compared with LPS, there was an obvious reduction in the conglutination of LPS+H2 cells, a reduction in VCAM-1, ICAM-1, and E-selectin levels, and a reduction in the TEER-resistance value, while the expression of VE-cadherin increased. Fluorescence results showed that, compared with control cells, the VE-cadherin in LPS cells was in-complete at the cell joints. Compared with LPS cells, the VE-cadherin in LPS+H2 cells was even and complete at the cell joints. Liquid rich in hydrogen could reduce LPS-induced production of adhesion molecules and endothelium-hyaline leukocyte conglutination, and influence the expression and distribution of VE-cadherin to regulate the permeability of the endothelium. /Hydrogen-rich medium/Maternal inflammation is associated with spontaneous preterm birth and respiratory impairment among premature infants. Recently, molecular hydrogen (H2) has been reported to have a suppressive effect on oxidative stress and inflammation. The aim of this study was to evaluate the effects of H2 on fetal lung injury caused by maternal inflammation. Cell viability and the production of interleukin-6 (IL-6) and reactive oxygen species (ROS) were examined by treatment with lipopolysaccharide (LPS) contained in ordinal or H2-rich medium (HM) using a human lung epithelial cell line, A549. Pregnant Sprague Dawley rats were divided into three groups: Control, LPS, and HW + LPS groups. Rats were injected with phosphate-buffered saline (Control) or LPS intraperitoneally (LPS) on gestational day 19 and provided H2 water (HW) ad libitum for 24 hr before LPS injection (HW + LPS). Fetal lung samples were collected on day 20, and the levels of apoptosis, oxidative damage, IL-6, and vascular endothelial growth factor (VEGF) were evaluated using immunohistochemistry. The number of apoptotic cells, and levels of ROS and IL-6 were significantly increased by LPS treatment, and repressed following cultured with HM in A549 cells. In the rat models, the population positive for cleaved caspase-3, 8-hydroxy-2'-deoxyguanosine, IL-6, and VEGF was significantly increased in the LPS group compared with that observed in the Control group and significantly decreased in the HW + LPS group. In this study, LPS administration induced apoptosis and oxidative damage in fetal lung cells that was ameliorated by maternal H2 intake. Antenatal H2 administration may decrease the pulmonary mobility associated with inflammation in premature infants. /Hydrogen-rich medium/An anesthetized rat preparation was used for breathing studies in hyperbaric H2/CO2 atmospheres in which total gaseous pressure was varied in the safe pressure range 8.3-19.1 atm. PCO2 (partial pressure of CO2) was studied at levels of 0.05 and 0.10 atm. The response variables measured were respiration rate, blood pH and an isotonic index of diaphragm twitch amplitude, in the presence of He/O2 gas mixtures used for compression and H2/O2/Co2 saturation gases. The rat preparation can be well maintained in H2/O2 environments at pressures in excess of 8 atm. Elevated PCO2 in these environments can evoke rapid increases in respiration rate and twitch amplitude, without any marked changes in blood pH. Graded increases in PH2 (partial pressure of H2) are able to lower and ultimately abolish the effects of either 0.05 or 0.10 atm levels of CO2 in breathing parameters. The intrinsic ability of hyperbaric H2 to effect a CO2-sparing action on this preparation is greater than that previously seen with hyperbaric Ar or He. The potency sequence for the sparing action by the inert component of the hyperbaric breathing gas is H2 > Ar > He.For more Interactions (Complete) data for Hydrogen (30 total), please visit the HSDB record page. |
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| 유해 작용 | Other Poison - Simple Asphyxiant |
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| 노출 경로 | Exposure mainly occurs via inhalation. |
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| 독성 요약 | IDENTIFICATION AND USE: Hydrogen is a colorless gas. Its many uses include the following: production of ammonia, ethanol, and anilinehydrocracking, hydroforming, and hydrofining of petroleumhydrogenation of vegetable oilshydrogenolysis of coalreducing agent for organic synthesis and metallic oresreducing atmosphere to prevent oxidationas oxyhydrogen flame for high temperaturesatomic-hydrogen weldinginstrument-carrying balloonsmaking hydrogen chloride and hydrogen bromideproduction of high-purity metalsfuel for nuclear rocket engines for hypersonic transportmissile fuelcryogenic research. In addition, hydrogen is a versatile energy carrier that can be used to power nearly every end-use energy need (Fuel cells). Molecular hydrogen (H2) emerged as a novel therapeutic agent, with antioxidant, anti-inflammatory and anti-apoptotic effects demonstrated in plethora of animal disease models and human studies. HUMAN STUDIES: Hydrogen is a simple asphyxiant. Contact with liquid hydrogen will cause frostbite or severe burns of the skin. Hydrogen-rich water has been tested for treating oxidative stress-induced disorders because of its reactive oxygen species scavenging abilities. Hydrogen therapy may be an effective and specific innovative treatment for exercise-induced oxidative stress and sports injury, with potential for the improvement of exercise performance. ANIMAL STUDIES: A large bubble of the gas injected into anterior chamber of rabbit eyes was absorbed within three days and caused no injury. H2 was believed to be inert and nonfunctional in mammalian cells. More recently it was demonstrated that H2 reacts with highly reactive oxidants such as hydroxyl radical and peroxynitrite inside cells. Beneficial effects of molecular hydrogen in animal models were observed especially in oxidative stress-mediated diseases, such as diabetes mellitus, brain stem infarction, rheumatoid arthritis, or neurodegenerative diseases. H2 affects cell signal transduction. |
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| Signs and Symptoms | Dizziness. Headache. Lethargy. Suffocation.ON CONTACT WITH GAS: FROSTBITE. |
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| Natural Pollution Sources | ... Hydrogen is the most abundant of all elements in the universe, and it is thought that the heavier elements were, and still are, being built from hydrogen and helium. It has been estimated that hydrogen makes up more than 90% of all the atoms or three quarters of the mass of the universe. It is found in the sun and most stars, and plays an important part in the proton-proton reaction and carbon-nitrogen cycle, which accounts for the energy of the sun and stars. It is thought that hydrogen is a major component of the planet Jupiter and that at some depth in the planet's interior the pressure is so great that solid hydrogen is converted into solid metallic hydrogen.Occurrence in earth's atmosphere 0.00005% H2.Hydrogen from natural sources consists of more than 99.8% protium. ... |
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| Human Toxicity Excerpts | /SIGNS AND SYMPTOMS/ No specific toxic action. In high concn can act as a simple asphyxiant./SIGNS AND SYMPTOMS/ Contact with cryogenic liquid hydrogen will cause frostbite or severe burns of the skin. Simple asphyxiant./SIGNS AND SYMPTOMS/ The relation between the /CNS depressant/ effect of nitrogen and that of hydrogen is 1:0.26./SIGNS AND SYMPTOMS/ Hydrogen has no known toxic effect on the eye.For more Human Toxicity Excerpts (Complete) data for Hydrogen (11 total), please visit the HSDB record page. |
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| Non-Human Toxicity Excerpts | /LABORATORY ANIMALS: Acute Exposure/ The nuclear protein high-mobility group box 1 (HMGB1) is a key trigger for the inflammatory reaction during liver ischemia reperfusion injury (IRI). Hydrogen treatment was recently associated with down-regulation of the expression of HMGB1 and pro-inflammatory cytokines during sepsis and myocardial IRI, but it is not known whether hydrogen has an effect on HMGB1 in liver IRI. A rat model of 60 minutes 70% partial liver ischemia reperfusion injury was used. Hydrogen enriched saline (2.5, 5 or 10 mL/kg) was injected intraperitoneally 10 minutes before hepatic reperfusion. Liver injury was assessed by serum alanine aminotransferase (ALT) enzyme levels and histological changes. We also measured malondialdehyde (MDA), hydroxynonenal (HNE) and 8-hydroxy-guanosine (8-OH-G) levels as markers of the peroxidation injury induced by reactive oxygen species (ROS). In addition, pro-inflammatory cytokines including TNF-a and IL-6, and high mobility group box B1 protein (HMGB1) were measured as markers of post ischemia-reperfusion inflammation. Hydrogen enriched saline treatment significantly attenuated the severity of liver injury induced by ischemia-reperfusion. The treatment group showed reduced serum ALT activity and markers of lipid peroxidation and post ischemia reperfusion histological changes were reduced. Hydrogen enriched saline treatment inhibited HMGB1 expression and release, reflecting a reduced local and systemic inflammatory response to hepatic ischemia reperfusion. These results suggest that, in our model, hydrogen enriched saline treatment is protective against liver ischemia-reperfusion injury. This effect may be mediated by both the anti-oxidative and anti-inflammatory effects of the solution. /Hydrogen enriched saline//LABORATORY ANIMALS: Subchronic or Prechronic Exposure/ Hydrogen has been reported to relieve damage in many disease models, and is a potential additive in drinking water to provide protective effects for patients as several clinical studies revealed. However, the absence of a dose-response relationship in the application of hydrogen is puzzling. We attempted to identify the dose-response relationship of hydrogen in alkaline electrolyzed drinking water through the aspirin induced gastric injury model. In this study, hydrogen-rich alkaline water was obtained by adding H2 to electrolyzed water at one atmosphere pressure. After 2 weeks of drinking, we detected the gastric mucosal damage together with MPO, MDA and 8-OHdG in rat aspirin induced gastric injury model. Hydrogen-dose dependent inhibition was observed in stomach mucosal. Under pH 8.5, 0.07, 0.22 and 0.84 ppm hydrogen exhibited a high correlation with inhibitory effects showed by erosion area, MPO activity and MDA content in the stomach. Gastric histology also demonstrated the inhibition of damage by hydrogen-rich alkaline water. However, 8-OHdG level in serum did not have significant hydrogen-dose dependent effect. pH 9.5 showed higher but not significant inhibitory response compared with pH 8.5. Hydrogen is effective in relieving the gastric injury induced by aspirin-HCl, and the inhibitory effect is dose-dependent. The reason behind this may be that hydrogen-rich water directly interacted with the target tissue, while the hydrogen concentration in blood was buffered by liver glycogen, evoking a suppressed dose-response effect. Drinking hydrogen-rich water may protect healthy individuals from gastric damage caused by oxidative stress. /Hydrogen water//LABORATORY ANIMALS: Subchronic or Prechronic Exposure/ The prevalence of sleep apnea is very high in patients with heart failure (HF). The aims of this study were to investigate the influence of intermittent hypoxia (IH) on the failing heart and to evaluate the antioxidant effect of hydrogen gas. Normal male Syrian hamsters (n = 22) and cardiomyopathic (CM) hamsters (n = 33) were exposed to IH (repeated cycles of 1.5 min of 5% oxygen and 5 min of 21% oxygen for 8 hr during the daytime) or normoxia for 14 days. Hydrogen gas (3.05 vol/100 vol) was inhaled by some CM hamsters during hypoxia. IH increased the ratio of early diastolic mitral inflow velocity to mitral annulus velocity (E/e', 21.8 vs. 16.9) but did not affect the LV ejection fraction (EF) in normal Syrian hamsters. However, IH increased E/e' (29.4 vs. 21.5) and significantly decreased the EF (37.2 vs. 47.2%) in CM hamsters. IH also increased the cardiomyocyte cross-sectional area (672 vs. 443 sq um) and interstitial fibrosis (29.9 vs. 9.6%), along with elevation of oxidative stress and superoxide production in the left ventricular (LV) myocardium. Furthermore, IH significantly increased the expression of brain natriuretic peptide, beta-myosin heavy chain, c-fos, and c-jun mRNA in CM hamsters. Hydrogen gas inhalation significantly decreased both oxidative stress and embryonic gene expression, thus preserving cardiac function in CM hamsters. In conclusion, IH accelerated LV remodeling in CM hamsters, at least partly by increasing oxidative stress in the failing heart. These findings might explain the poor prognosis of patients with HF and sleep apnea./LABORATORY ANIMALS: Developmental or Reproductive Toxicity/ Maternal inflammation is associated with spontaneous preterm birth and respiratory impairment among premature infants. Recently, molecular hydrogen (H2) has been reported to have a suppressive effect on oxidative stress and inflammation. The aim of this study was to evaluate the effects of H2 on fetal lung injury caused by maternal inflammation. Cell viability and the production of interleukin-6 (IL-6) and reactive oxygen species (ROS) were examined by treatment with lipopolysaccharide (LPS) contained in ordinal or H2-rich medium (HM) using a human lung epithelial cell line, A549. Pregnant Sprague Dawley rats were divided into three groups: Control, LPS, and HW + LPS groups. Rats were injected with phosphate-buffered saline (Control) or LPS intraperitoneally (LPS) on gestational day 19 and provided H2 water (HW) ad libitum for 24 hr before LPS injection (HW + LPS). Fetal lung samples were collected on day 20, and the levels of apoptosis, oxidative damage, IL-6, and vascular endothelial growth factor (VEGF) were evaluated using immunohistochemistry. The number of apoptotic cells, and levels of ROS and IL-6 were significantly increased by LPS treatment, and repressed following cultured with HM in A549 cells. In the rat models, the population positive for cleaved caspase-3, 8-hydroxy-2'-deoxyguanosine, IL-6, and VEGF was significantly increased in the LPS group compared with that observed in the Control group and significantly decreased in the HW + LPS group. In this study, LPS administration induced apoptosis and oxidative damage in fetal lung cells that was ameliorated by maternal H2 intake. Antenatal H2 administration may decrease the pulmonary mobility associated with inflammation in premature infants. /Hydrogen-rich medium/For more Non-Human Toxicity Excerpts (Complete) data for Hydrogen (16 total), please visit the HSDB record page. |
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| Probable Routes of Human Exposure | According to the 2016 TSCA Inventory Update Reporting data, nn reporting facilities estimate the number of persons reasonably likely to be exposed during the manufacturing, processing, or use of hydrogen in the United States may be as low as <10 workers and as high as <10,000 workers per plantthe data may be greatly underestimated due to confidential business information (CBI) or unknown values(1). |
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| Antidote and Emergency Treatment | Immediate first aid: Ensure that adequate decontamination has been carried out. If patient is not breathing, start artificial respiration, preferably with a demand-valve resuscitator, bag-valve-mask device, or pocket mask, as trained. Perform CPR as necessary. Immediately flush contaminated eyes with gently flowing water. Do not induce vomiting. If vomiting occurs, lean patient forward or place on left side (head-down position, if possible) to maintain an open airway and prevent aspiration. Keep patient quiet and maintain normal body temperature. Obtain medical attention. /Flammable gases/Basic treatment: Establish a patent airway (oropharyngeal or nasopharyngeal airway, if needed). Suction if necessary. Watch for signs of respiratory insufficiency and assist ventilations as necessary. Administer oxygen by nonrebreather mask at 10 to 15 L/min. Monitor for pulmonary edema and treat if necessary ... . Monitor for shock and treat if necessary ... . Anticipate seizures and treat if necessary ... . For eye contamination, flush eyes immediately with water. Irrigate each eye continuously with 0.9% saline (NS) during transport ... . Treat frostbite with rapid rewarming ... . /Flammable gases/Advanced treatment: Consider orotracheal or nasotracheal intubation for airway control in the patient who is unconscious, has severe pulmonary edema, or is in severe respiratory distress. Positive-pressure ventilation techniques with a bag valve mask device may be beneficial. Consider drug therapy for pulmonary edema ... . Monitor cardiac rhythm and treat arrhythmias as necessary ... . Start IV administration of D5W /SRP: "To keep open", minimal flow rate/. Use 0.9% saline (NS) or lactated Ringer's (LR) if signs of hypovolemia are present. For hypotension with signs of hypovolemia, administer fluid cautiously. Watch for signs of fluid overload ... . Treat seizures with diazepam or lorazepam ... . Use proparacaine hydrochloride to assist eye irrigation ... . /Flammable gases/Immediate first aid: Ensure that adequate decontamination has been carried out. If patient is not breathing, start artificial respiration, preferably with a demand-valve resuscitator, bag-valve-mask device, or pocket mask, as trained. Perform CPR as necessary. Immediately flush contaminated eyes with gently flowing water. Do not induce vomiting. If vomiting occurs, lean patient forward or place on left side (head-down position, if possible) to maintain an open airway and prevent aspiration. Keep patient quiet and maintain normal body temperature. Obtain medical attention. /Simple asphyxiants and related compounds/For more Antidote and Emergency Treatment (Complete) data for Hydrogen (6 total), please visit the HSDB record page. |
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| DHS Chemicals of Interest (COI) | Hydrogen110000Flammable chemical that can be released at a facility. |
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| 신호 어 | Danger |
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| 유해·위험 문구 | 암호 유해·위험 문구 위험 등급 범주 신호 어 그림 문자 P- 코드 H220 |
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