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HCID 2513Erstellt: 31. Oktober 2026

Dimethylarsinsure

DIMETHYLARSINIC ACID · DIMETHYLARSONIC ACID · Ansar · Arsan · Dilic · Erase · Salvo · Phytar

https://huagongshe.com/de/chemical/2513

Molekülstruktur von Dimethylarsinsure

Identifikatoren & physikalische Daten

CAS No.75-60-5
SummenformelC2H7AsO2
Durchschnittliche Molmasse138 g/mol
Monoisotopische Masse137,966199 g/mol
SMILESC[As](C)(=O)O
InChIKeyOGGXGZAMXPVRFZ-UHFFFAOYSA-N
PubChem CID2513
ChEMBLCHEMBL1231644
ChEBICHEBI:48765
EC No.200-883-4
DTXSIDDTXSID7020508
Lokaler NameDimethylarsinsure

Chemische & physikalische Eigenschaften

Polarare Oberfläche (TPSA)37,3
H-Brücken-Donoren1
H-Brücken-Akzeptoren2
Frei drehbare Bindungen0
Schwere Atome5
Formalladung0
Komplexität62
StoffklassenMetals -> Arsenic Compounds, OrganicCarcinogens
PestizidePesticide -> EPA IRIS
GeruchOdorless
Dichtegreater than 1.1 at 68 °F (est.) (USCG, 1999)
Color/FormCrystals from alcohol and etherColorlessTRICLINIC CRYSTALSWhitewater solutions may be dyed blue
LöslichkeitVery soluble (NTP, 1992)Soluble in acetic acidSoluble in ethanolinsoluble in diethyl etherIn water, 2X10+6 mg/l @ 25 °C.Methanol (Slightly), Water (Slightly)Soluble in water (50 mg/ml), water (2000 at 25°C), alcohols, and acetic acid. Insoluble in ether.
CorrosivityAll formulations are mildly corrosive.
Siedepunktgreater than 392 °F at 760 mmHg (NTP, 1992)>200 °C
DecompositionWhen heated to decomposition it emits toxic fumes of /arsenics/.Decomposed by powerful oxidizing or reducing agents.
Schmelzpunkt383 to 385 °F (NTP, 1992)195 °C195-198 °C
Dampfdruck0.0000001 [mmHg]
Physical DescriptionCacodylic acid appears as a colorless, odorless crystalline solid. Melting point 195-196 °C. Toxic by ingestion and irritating to skin and eyes.Hygroscopic solid[Merck Index] Colorless odorless hygroscopic solid[CAMEO] White crystalline solid[MSDSonline]
LagerbedingungenStore at RT.
AggregatzustandPowder/Solid
pKa (Säurekonstante)1.57(at 25℃)
FarbeWhite
Merck14,1604
BRN1736965
StabilitätAqueous solutions react violently with active metals. Incompatible with strong oxidizing agents, strong bases.
InChI1S/C2H7AsO2/c1-3(2,4)5/h1-2H3,(H,4,5)
InChIKeyOGGXGZAMXPVRFZ-UHFFFAOYSA-N
SMILESC[As](C)(O)=O
CAS Datenbank75-60-5(CAS DataBase Reference)
EPA chemische InformationenCacodylic acid (75-60-5)

Sicherheit & Vorschriften

GHS-EinstufungDangerH301 (94.2%): Toxic if swallowed [Danger Acute toxicity, oral]P261, P264, P270, P271, P273, P301+P316, P304+P340, P316, P321, P330, P391, P403+P233, P405, and P501 (click each P-code to see the statement)Aggregated GHS information provided per 52 reports by companies from 7 notifications to the ECHA C&L Inventory. Each notification may be associated with multiple companies.
Regulatorische InformationenHazard Traits - CarcinogenicityCardiovascular ToxicityDermatotoxicityDevelopmental ToxicityNeurotoxicityOther Toxicological Hazard TraitsRespiratory ToxicityRestricted substance: Dimethylarsinic acidArsinic acid, dimethyl-: Does not have an individual approval but may be used under an appropriate group standardThe New Jersey Worker and Community Right to Know Act requires public and private employers to provide information about hazardous substances at their workplaces. (N.J.S.A. 34:5A-1 et. seq.)
Weitere SicherheitsinformationenIMAP assessments - Arsinic acid, dimethyl-: Environment tier I assessment
DOT-KennzeichnungPoison
BrandgefahrenBehavior in Fire: May form toxic oxides of arsenic when heated. (USCG, 1999)· Non-combustible, substance itself does not burn but may decompose upon heating to produce corrosive and/or toxic fumes.
BrandpotenzialNonflammable
GesundheitsgefahrenChemical is essentially non-irritating in contact with skin or eyes. Ingestion causes arsenic poisoning, but symptoms are delayed. (USCG, 1999)· Highly toxic, may be fatal if inhaled, ingested or absorbed through skin.
GefahrenübersichtThere is sufficient evidence in experimental animals for the carcinogenicity of dimethylarsenic acid. . . . Arsenic in drinking-water is carcinogenic to humans (Group 1). [IARC] A forestry worker injecting trees with silvicide containing cacodylic acid developed anorexia, nausea, and abdominal pain

Anwendungen & Verwendung

Verwenden

Cacodylic acid is used for dermatologic treatment in cronic eczema, anemia and as a tonic.

He had elevated urine arsenic, and symptoms subsided after termination of exposure
[HSDB] Essentially non-irritating in contact with skin and eyes
[CHRIS] Not converted into inorganic arsenic in the body
[Nordberg, p. 590] Most excreted unchanged
[Rosenstock, p. 947]
DOT-Nummer und Handbuch1572 151
Reaktive GruppeAcids, Carboxylic
RCRA-AnforderungenU136As stipulated in 40 CFR 261.33, when cacodylic acid, as a commercial chemical product or manufacturing chemical intermediate or an off-specification commercial chemical product or a manufacturing chemical intermediate, becomes a waste, it must be managed according to Federal and/or State hazardous waste regulations. Also defined as a hazardous waste is any residue, contaminated soil, water, or other debris resulting from the cleanup of a spill, into water or on dry land, of this waste. Generators of small quantities of this waste may qualify for partial exclusion from hazardous waste regulations (40 CFR 261.5).
FIFRA-AnforderungenAs the federal pesticide law FIFRA directs, EPA is conducting a comprehensive review of older pesticides to consider their health and environmental effects and make decisions about their future use. Under this pesticide reregistration program, EPA examines health and safety data for pesticide active ingredients initially registered before November 1, 1984, and determines whether they are eligible for reregistration. In addition, all pesticides must meet the new safety standard of the Food Quality Protection Act of 1996. Pesticides for which EPA had not issued Registration Standards prior to the effective date of FIFRA, as amended in 1988, were divided into three lists based upon their potential for human exposure and other factors, with List B containing pesticides of greater concern and List D pesticides of less concern. Cacodylic acid is found on List B. Case No: 2080Pesticide type: HerbicideCase Status: OPP is reviewing data from the pesticide's producers regarding its human health and/or environmental effects, or OPP is determining the pesticide's eligibility for reregistration and developing the Reregistration Eligibility Decision (RED) document.Active ingredient (AI): cacodylic acidData Call-in (DCI) Date(s): 06/07/91, 03/03/95, 10/13/95AI Status: The producers of the pesticide has made commitments to conduct the studies and pay the fees required for reregistration, and are meeting those commitments in a timely manner.Tolerances are established for residues of the defoliant cacodylic acid (dimethylarsenic acid), expressed as As2O3, in or on raw agricultural commodities as follows: in or on cottonseedin the kidney and liver of cattleand in meat, fat, and meat byproducts (except kidney and liver) of cattle.If material involved in fire: Extinguish fire using agent suitable for type of surrounding fire. (Material itself does not burn or burns with difficulty.) Use water in flooding quantities as fog. Use foam, dry chemical, or carbon dioxide. /Arsenical cmpd, liquid, NOS/If material involved in fire: Extinguish fire using agent suitable for type of surrounding fire. (Material itself does not burn or burns with difficulty.) Use water in flooding quantities as fog. Use foam, dry chemical, or carbon dioxide. /Arsenical cmpd, solid, NOS/
OSHA StandardsPermissible Exposure Limit: Table Z-1 8-Hr Time Weighted Avg: 0.5 mg/cu m. /Arsenic, organic cmpd (as As)/
BrandbekämpfungExcerpt from ERG Guide 151 [Substances - Toxic (Non-Combustible)]:
Freisetungsmaßnahmen· CALL 911. Then call emergency response telephone number on shipping paper. If shipping paper not available or no answer, refer to appropriate telephone number listed on the inside back cover.· Do not touch damaged containers or spilled material unless wearing appropriate protective clothing.
Erste HilfeEYES: First check the victim for contact lenses and remove if present. Flush victim's eyes with water or normal saline solution for 20 to 30 minutes while simultaneously calling a hospital or poison control center. Do not put any ointments, oils, or medication in the victim's eyes without specific instructions from a physician. IMMEDIATELY transport the victim after flushing eyes to a hospital even if no symptoms (such as redness or irritation) develop.General First Aid:
Expositionsschutz und Persönliche Schutzausrüstung· Wear positive pressure self-contained breathing apparatus (SCBA).
BrandbekämpfungsverfahrenIf material on fire or involved in fire: Extinguish fire using agent suitable for type of surrounding fire. (Material itself does not burn or burns with difficulty.) Use water in flooding quantities as fog. Use foam, dry chemical or carbon dioxide. Keep run-off water out of water sources and sewers.
LagerbedingungenKEEP WELL CLOSED.DO NOT STORE NEAR FERTILIZERS, SEEDS, INSECTICIDES, /OR/ FUNGICIDES.
Freisetung ohne BrandExcerpt from ERG Guide 151 [Substances - Toxic (Non-Combustible)]:
EntsorgungsverfahrenGenerators of waste (equal to or greater than 100 kg/mo) containing this contaminant, EPA hazardous waste number D004, must conform with USEPA regulations in storage, transportation, treatment and disposal of waste.Generators of waste (equal to or greater than 100 kg/mo) containing this contaminant, EPA hazardous waste number U136, must conform with USEPA regulations in storage, transportation, treatment and disposal of waste.Storage: To convert the gas-cleaning residues obtained during the metallurgical processing of arsenic-containing ores into a portable and less water-soluble form, the metals are precipitated as hydroxides by using an excess of lime water and the arsenic is precipitated as calcium arsenate and calcium arsenite. This "arsenic sludge" is recycled, on the one hand, in order not to lose the valuable metals, and on the other, in order to reduce the problem of arsenic sludge disposal.A poor candidate for incineration.
Preventive MeasuresIf material not on fire and not involved in fire: Keep sparks, flames, and other sources of ignition away. Keep material out of water sources and sewers.Personnel protection: Avoid breathing dusts, and fumes from burning material. Keep upwind. ... Avoid bodily contact with the material. ... Do not handle broken packages unless wearing appropriate personal protective equipment.
Isolation and EvacuationExcerpt from ERG Guide 151 [Substances - Toxic (Non-Combustible)]:Immediate precautionary measure
Minimum Risk LevelAcute Oral: 0.005 mg/kg/day (L134) Chronic Oral: 0.0003 mg/kg/day (L134) Chronic Inhalation: 0.01 mg/m3 (L134)
Allowable TolerancesTolerances are established for residues of the defoliant cacodylic acid (dimethylarsenic acid), expressed as As2O3, in or on raw agricultural commodities as follows: 2.8 ppm in or on cottonseed1.4 ppm in the kidney and liver of cattleand 0.7 ppm in meat, fat, and meat byproducts (except kidney and liver) of cattle.Tolerances for total residues of combined arsenic (calculated as As) in food are established as follows: (a) In edible tissues & in eggs of chickens & turkeys: 0.5 ppm in uncooked muscle tissue2 ppm in uncooked edible by-products& 0.5 ppm in eggs. (b) In edible tissues of swine: 2 ppm in uncooked liver & kidney0.5 ppm in uncooked muscle tissue & by-products other than liver & kidney. /Arsenic/
KörperbelastungUrinary excretion of arsenic has been directly correlated with worker exposure to dimethylarsenic acid(1,2). Blood levels of arsenic does not seem to be correlated with worker exposure to dimethylarsenic acid(1). Herbicide workers using poor handling and application techniques had urine arsenic levels as high as 1.8 ppm which corresponded to an exposure of at least 0.036 mg As/kg body wt/day(2)proper handling techniques and protective gear reduced exposure by an order of magnitude(2). In forestry workers, the urinary dimethylarsenic acid level during an 11 week observation period ranged from 24-172 ug/24 hr compared to a range of 26-73 ug/24 hr for non-exposed workers(1). Urine samples of humans were found to contain an average of 15 ug/l of dimethylarsenic acid which constituted an average of 66% of the total urinary excretion of arsenic(3).
TreatmentArsenic poisoning can be treated by chelation therapy, using chelating agents such as dimercaprol, EDTA or DMSA. Charcoal tablets may also be used for less severe cases. In addition, maintaining a diet high in sulfur helps eliminate arsenic from the body. (L20)Aggressive decontamination with gastric lavage is recommended if the patient has consumed a potentially life-threatening dose and if the patient is not vomiting. Administer charcoal as a slurry (T36).
WechselwirkungenThe influence of the methyltransferase inhibitor periodate oxidized adenosine on the metabolism of arsenite was investigated in mice and rabbits. Groups of male NMRI-mice and New Zealand rabbits were given intraperitoneal (ip) injections of 100 um periodate oxidized adenosine per kilogram (kg) 15 min before an intravenous injection of 0.04 mg/kg (74)As labeled arsenite or injections of (74)As labeled arsenite only. These animals were kept for 16 to 72 hr in metabolic cages designed to separate urine and feces. The urinary concn of dimethylarsenic acid (DMA), the major arsenic metabolite, was measured. Other groups of animals were injected with (74)As labeled arsenite and periodate oxidized adenosine or (74)As labeled arsenite only and killed between 1 to 72 hr. Blood, liver, kidney, lung, epididymis, and portions of skin were removed and measured for (74)As content in a gamma scintillation counter. Also, subcellular fractionation of liver tissue was performed and the concentration of (74)As was ascertained. The urinary excretion of (74)As was lower in mice and rabbits treated with periodate oxidized adenosine prior to arsenite administration than in animals treated with arsenite only. Injection of periodate oxidized adenosine prior to arsenite produced a 25 to 70% decrease in the production of dimethylarsenic acid, implying that S-adenosylmethionine was the methyl donor for the methylation of inorganic arsenic in vivo. The fecal excretion of (74)As was less than 4% of the dose, independent of treatment and animal species. Periodate oxidized adenosine treated animals had 2 to 6 times higher concentrations of (74)As in tissues than that in controls, the effect being first observed in liver tissues. The subcellular distribution of (74)As in liver of mice was not affected by periodate oxidized adenosine treatment in that 50% was found in the soluble cytoplasmic fraction and 20 to 30% was found in thenuclear fraction, independent of the treatment.
Gesundheitliche AuswirkungenArsenic poisoning can lead to death from multi-system organ failure, probably from necrotic cell death, not apoptosis. Arsenic is also a known carcinogen, especially in skin, liver, bladder and lung cancers. (T1, L20)Hypovolemia from capillary leakage (third-spacing of fluids) is a common, serious, early effect. Muscle cramps, facial edema, bronchitis, dyspnea, chest pain, dehydration, intense thirst, and fluid-electrolyte disturbances are also common following significant exposures. A garlic-like odor of the breath and feces may also occur. After absorption, arsenic may cause multi-organ failure by inhibiting sulfhydryl-containing enzymes. Encephalopathy, with headache, lethargy, mental confusion, hallucinations, emotional lability, memory loss and delirium may occurseizures, stupor, convulsions, coma, and death may follow within 24 hours of a severe acute exposure. Dysrhythmias (particularly QTc prolongation and torsade de pointes), cardiomyopathy, ARDS, hepatitis, rhabdomyolysis, hemolysis, and renal failure may develop over several days. The sequence of chronic poisoning involves hyperpigmentation, and eczematoid and allergic dermatitis. muscle fasciculationsgross tremorsataxiaincoordinationand mental confusion. Muscular weakness, limb tenderness and difficulty walking may follow. The final phase consists of peripheral sensory neuropathy of the hands and feet. That may be associated with a motor neuropathy as well (T36).
UmweltverhaltenTERRESTRIAL FATE: ... /CACODYLIC ACID/ REACTS /IN SOIL/ WITH TRIVALENT CHROMIUM, SILVER NITRATE & OTHER METAL IONS ... MAY FORM CYCLIC TYPES OF CMPD UNDER CERTAIN CONDITIONS.TERRESTRIAL FATE: CHEM & BIOLOGICAL TRANSFORMATION OF ARSENICAL IN SOIL: IN SOIL CACODYLIC ACID MAY UNDERGO SALT FORMATION, ADSORPTION, ION EXCHANGE, DEMETHYLATION (OXIDATIVE), REDN, & METHYLATION (REDUCTIVE). /FROM TABLE/TERRESTRIAL FATE: CACODYLIC ACID FORMED INSOL CMPD WITH SOIL.TERRESTRIAL FATE: Dimethylarsenic acid is expected to have low mobility in soils(1-4). Organoarsenicals, such as dimethylarsinic acid, are adsorbed by clays soils(1). After rapid initial adsorption, changes occur which result in the redistribution of dimethylarsinic acid into a less soluble form associated with aluminum in the soil(1). The dimethylarsinic acid is fixed by iron and aluminum in the soil, although not as strongly as inorganic arsenate(1). Arsenic residues in browse and herbaceous vegetation in Douglas fir, ponderosa pine, and western larch forests in which trees were treated with cacodylic acid, sodium salt for thinning were relatively low(1). Large quantities of arsenic did not move from the forest floor into the soil, indicating that arsenic residues were tightly bound in the foliage(1). Only small amounts of arsenic were detected in streams in forests treated with cacodylic acid or its sodium salt(1). A pKa of 1.57(5) indicates that dimethylarsenic acid will ionize in water(SRC)therefore, dimethylarsenic acid is expected to be essentially nonvolatile from moist soil surfaces(SRC). Dimethylarsenic acid is not expected to volatilize from dry soil surfaces(SRC) based upon an estimated vapor pressure of 4.6X10-3 mm Hg(SRC), determined from a fragment constant method(6). Generation of C-14 labeled CO2 from C-14 labeled dimethylarsenic acid in a clay loam soil reached about 13% (over 98 days of incubation) in soil that had been adapted to dimethylarsenic acid, but was less than 2% in unadapted soil(7)the degradation was presumably microbial in nature(7). Incubation of dimethylarsenic acid in two Japanese soils for a period of six weeks resulted in 4-43% degradation(8)0% degradation occurred in sterile controls(8). In another study in soil, 36% of dimethylarsenic acid was converted to a volatile organoarsenical compound and 47% to carbon dioxide and arsenate within a 24-week period(9).For more Environmental Fate (Complete) data for DIMETHYLARSENIC ACID (6 total), please visit the HSDB record page.
Unerwünschte WirkungenIARC Carcinogen - Class 3: Chemicals are not classifiable by the International Agency for Research on Cancer.
ExpositionspfadeOral (L2)inhalation (L2)dermal (L2)
ToxizitätsübersichtArsenic and its metabolites disrupt ATP production through different mechanisms. At the level of the citric acid cycle, arsenic inhibits the pyruvate dehydrogenase and uncouples the oxidative phosphorylation by competing with phosphate. This leads to inhibition of energy-linked reduction of NAD+, mitochondrial respiration, and ATP synthesis. Hydrogen peroxide production is increased, leading to oxidative stress due to the formation of reactive oxygen species. Arsenic's carginogenicity is influenced by the arsenical binding of tubulin, which results in aneuploidy, polyploidy and mitotic arrests. The binding of other arsenic protein targets may also cause altered DNA repair enzyme activity, altered DNA methylation patterns and cell proliferation. (T1, A17)Arsenic and its metabolites disrupt ATP production through several mechanisms. At the level of the citric acid cycle, arsenic inhibits pyruvate dehydrogenase and by competing with phosphate it uncouples oxidative phosphorylation, thus inhibiting energy-linked reduction of NAD+, mitochondrial respiration, and ATP synthesis. Hydrogen peroxide production is also increased, which might form reactive oxygen species and oxidative stress. Arsenic's carcinogenicity is influenced by the arsenical binding of tubulin, which results in aneuploidy, polyploidy and mitotic arrests. The binding of other arsenic protein targets may also cause altered DNA repair enzyme activity, altered DNA methylation patterns and cell proliferation. (T1, A17)
Plant ConcentrationsTHE DEFOLIANT FORMULATION BOLLS-EYE, WHICH CONTAINS CACODYLIC ACID & ITS SODIUM SALT, & INORGANIC ARSENIC WAS SPRAYED ON COTTON & RESIDUES WERE DETERMINED 2, 4, 8, & 25 DAYS AFTER APPLICATION. TOTAL ARSENIC, METHANEARSINIC ACID & TOTAL CACODYLICS & ARSENATE PEAKED IMMEDIATELY AFTER SPRAYING & DECLINED. BY DAY 8 THE LEVELS WERE NOT SIGNIFICANTLY ABOVE PRE-APPLICATION BACKGROUND LEVELS. COTTON SEED (TOTAL ARSENIC) LEVELS WERE LOW, SUGGESTING NO DETECTABLE TRANSPORT.
Signs and SymptomsExposure to lower levels of arsenic can cause nausea and vomiting, decreased production of red and white blood cells, abnormal heart rhythm, damage to blood vessels, and a sensation of “pins and needles” in hands and feet. Breathing high levels of inorganic arsenic can provoque sore throat or irritated lungs. Arsenic also affects the brain, causing neurological disturbances such as headaches, confusion, and drowsiness. (A1)Initial signs and symptoms of arsenic ingestion include burning lips, throat constriction and dysphagia, followed by excruciating abdominal pain, hemorrhagic gastritis, gastroenteritis, severe nausea, projectile voting, profuse "rice water-like" diarrhea, with hypovolemia that may result in hypotension and an irregular pulse. The sequence of chronic poisoning involves weakness, anorexia, hepatomegaly, jaundice, and gastrointestinal complaints, followed by conjunctivitis, irritation of the throat and respiratory tract. ther effects of chronic exposure include conjunctivitis with irritation and lacrimationhair, skin and nail changeshyperkeratosis of feet and handsand melanosis, with pigment spots in corneal and conjunctival epithelium. Peripheral nervous system symptoms may include numbness, burning, and tingling of the hands and feetparesthesiastenderness (T36).
EPA IRIS InformationCacodylic acid
Soil Adsorption/MobilityOrganoarsenicals, such as dimethylarsinic acid, are adsorbed by clays soils(1). After rapid initial adsorption, changes occur which result in the redistribution of dimethylarsinic acid into a less soluble form associated with aluminum in the soil(1). The dimethylarsinic acid are fixed by iron and aluminum in the soil, although not as strongly as inorganic arsenate(1). Leaching tests conducted in specially constructed boxes with clay, silt loam, and sandy soils noted strong adsorption to all soils, although a small degree of leaching did occur(2). The adsorption of dimethylarsenic acid to sediments and soil was found to depend on clay content, iron oxide content, and pH(3,4)adsorption increases with increasing clay and iron oxide content and with higher pH(3,4). Herbicidal applications of dimethylarsenic acid that were applied to forest floors in the northwestern US were found to be tightly bound, and did not leach in soil(5).
LuftstandardsListed as a hazardous air pollutant (HAP) generally known or suspected to cause serious health problems. The Clean Air Act, as amended in 1990, directs EPA to set standards requiring major sources to sharply reduce routine emissions of toxic pollutants. EPA is required to establish and phase in specific performance based standards for all air emission sources that emit one or more of the listed pollutants. Dimethylarsenic acid is included on this list.(a) The owner or operator of an existing glass melting furnace subject to the provisions of this subpart shall comply with either paragraph (a)(1) or (a)(2) of this section ... (1) Uncontrolled total arsenic emissions from the glass melting furnace shall be less than 2.5 Mg (2.7 ton) per year, or ... (2) Total arsenic emissions from glass melting furnace shall be conveyed to a control device and reduced by at least 85%. /Total arsenic/(b) The owner or operator of a new or modified glass melting furnace subject to the provisions of this subpart shall comply with either paragraph (b)(1) or (b)(2) of this section ... (1) Uncontrolled total arsenic emissions from the glass melting furnace shall be less than 0.4 Mg (0.44 ton) per year, or ... (2) Total arsenic emissions from glass melting furnace shall be conveyed to a control device and reduced by at least 85%. /Total arsenic/The owner or operator of each copper converter subject to the provisions of this subpart shall reduce inorganic arsenic emissions to the atmosphere by meeting the following design, equipment, work practice, and operational requirements: (1) Install, operate, and maintain a secondary hood system on each copper converter. Each secondary hood system shall consist of a hood enclosure, air curtain fan(s), exhaust system fan(s), and ductwork that conveys the captured emission to a control device ... (2) Optimize the capture of secondary inorganic arsenic emission by operating the copper converter and secondary hood systems at all times ... . /Inorganic arsenic/
CERCLA-MeldepflichtmengenPersons in charge of vessels or facilities are required to notify the National Response Center (NRC) immediately, when there is a release of this designated hazardous substance, in an amount equal to or greater than its reportable quantity of 1 lb or 0.454 kg. The toll free number of the NRC is (800) 424-8802In the Washington D.C. metropolitan area (202) 426-2675. The rule for determining when notification is required is stated in 40 CFR 302.4 (section IV. D.3.b).
Clean Water Act AnforderungenToxic pollutant designated pursuant to section 307(a)(1) of the Federal Water Pollution Control Act and is subject to effluent limitations. /Arsenic & cmpd/For the maximum protection of human health from the potential carcinogenic effects due to exposure of arsenic through ingestion of contaminated water & contaminated aquatic organisms, the ambient water concn should be zero based on the nonthreshold assumption for this chemical. However, zero level may not be attainable at the present time. Therefore, the levels which may result in incremental increase of cancer risk over the lifetime are estimated at 1X10-5, 1X10-6, & 1X10-7. The corresponding criteria are 22 ng/l, 2.2 ng/l, & .22 ng/l, respectively .... For consumption of aquatic organisms only, excluding consumption of water, the levels are 175 ng/ml, 17.5 ng/l, & 1.75 ng/l, respectively. /Arsenic/D004A solid waste containing arsenic may or may not become characterized as a hazardous waste when subjected to the Toxicity Characteristic Leaching Procedure listed in 40 CFR 261.24, and if so characterized, must be managed as a hazardous waste.
Landesrichtlinien für Trinkwasser(AZ) ARIZONA 10 ug/L /Arsenic/(CT) CONNECTICUT 10 ug/l /Arsenic/
Federal Drinking Water Standards10 ug/l /Arsenic/
F3-10
Wassergefährdungsklasse (WGK)3
TSCA-Inventar-StatusTSCA listed
UN-TransportnummerUN 1572
Zollcode29310099
RTECS-Nr.CH7525000
R-Sätze (R)23/25-50/53
ToxizitätLD50 orally in rats: 1350 mg/kg (Bailey, White)
GefahrensymboleT,N
Sicherheitshinweise (S)20/21-28-45-60-61
Gefahrenklasse6.1Acute Tox. 3 InhalationAcute Tox. 3 OralAquatic Acute 1Aquatic Chronic 1Carc. 1B
VerpackungsgruppeII
Lagerklasse6.1A - Combustible acute toxic Cat. 1 and 2very toxic hazardous materials
Gefahrstoffdaten75-60-5(Hazardous Substances Data)