V-9-M cholecystokinin nonapeptide
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Grundinformationen
- CAS
99291-20-0- Standard-SMILES
CSCC[C@H](NC(=O)[C@@H]1CCCN1C(=O)[C@H](CC(=O)O)NC(=O)[C@@H](NC(=O)[C@H](C)NC(=O)[C@H](CCC(=O)O)NC(=O)[C@@H](NC(=O)C1CCCN1C(=O)[C@@H](N)C(C)C)C(C)C)C(C)C)C(=O)O- InChIKey
AIKMAJWJXJPJNB-HNRKYVDVNA-N- Summenformel
- C42H69N9O14S
- Durchschnittliche Molmasse
- 956,12 g/mol
Namen und Bezeichner
- Bevorzugter Name
- V-9-M cholecystokinin nonapeptide
- Englischer Name
- V-9-M cholecystokinin nonapeptide
- Summenformel
- C42H69N9O14S
- Molmasse
- 956.12
Namen und Synonyme
Cholecystokinin Precursor (24-32) (rat) H-Val-Pro-Val-Glu-Ala-Val-Asp-Pro-Met-OHSmeglutideSemaglutide（Recombinant）Cholecystokinin PrecursorV-9-M cholecystokinin nonapeptideL-Valyl-L-prolyl-L-valyl-L-α-glutamyl-L-alanyl-L-valyl-L-α-aspartyl-L-prolyl-L-methionineL-Methionine,L-valyl-L-prolyl-L-valyl-L-a-glutamyl-L-alanyl-L-valyl-L-a-aspartyl-L-prolyl-L-Methionine, L-valyl-L-prolyl-L-valyl-L-α-glutamyl-L-alanyl-L-valyl-L-α-aspartyl-L-prolyl-Cholecystokinin precursor (24-32), also known as semaglutide, is a human yeast fermented substanceV 9 M|||Prepro CCK Fragment V-9-M|||Val-pro-val-glu-ala-val-asp-pro-met|||Cholecystokinin Precursor (24-32)|||V9M|||V-9-M
Datenbank-Bezeichner
- CAS
99291-20-0- PubChem CID
- 5489386
Eigenschaften
Physikochemische Eigenschaften
- Siedepunkt
- 1350.7 · 1350.7±65.0 °C(Predicted)
- Dichte
- 1.306 · 1.306±0.06 g/cm3(Predicted)
- pka
- 3.51 · 3.51±0.10(Predicted)
- Aggregatzustand
- Solid
- Farbe
- White to off-white
- InChIKey
- AIKMAJWJXJPJNB-HNRKYVDVNA-N
Chemie und Industrie
Anwendungen
BeschreibungV-9-M Cholecystokinin nonapeptide is a glucagon-like peptide receptor agonist, namely GLP-1 receptor agonist. Human GLP-1 is a peptide hormone secreted by intestinal L cells. Cholecystokinin Precursor (24-32), rat has apparent sedative actions and prevents experimental amnesisa in rats.
VerwendenV-9-M cholecystokinin nonapeptide belongs to the glucagon-like peptide-1 receptor agonist (GLP-1 receptor agonist) class of hypoglycemic drugs, which increase insulin secretion in a glucose-dependent manner and inhibit glucagon secretion , and can promote gastric emptying, centrally suppress appetite, reduce food intake, and then reduce glucose absorption to achieve the effect of lowering blood sugar.
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