| GHS-Einstufung | DangerH270: May cause or intensify fireoxidizer [Danger Oxidizing gases]P220, P244, P370+P376, and P403 (click each P-code to see the statement)This chemical does not meet GHS hazard criteria for 1% (13 of 1351) of reports.H270 (99%): May cause or intensify fire |
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| Regulatorische Informationen | Chemical: OxygenOxygen is listed on the EPA's Chemical Data Reporting (CDR) system. Manufacturers and importers of Oxygen are required to report information about their production and use of this chemical to the EPA under the Toxic Substances Control Act (TSCA). (40 eCFR Part 711)Oxygen: HSNO Approval: HSR001029 Approved with controlsThe New Jersey Worker and Community Right to Know Act requires public and private employers to provide information about hazardous substances at their workplaces. (N.J.S.A. 34:5A-1 et. seq.) |
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| Weitere Sicherheitsinformationen | IMAP assessments - Oxygen: Human health tier I assessment |
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| DOT-Kennzeichnung | Non-Flammable Gas Oxidizer |
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| Brandgefahren | Behavior in Fire: Increases intensity of any fire. Mixtures of liquid oxygen and any fuel are highly explosive. (USCG, 1999)Excerpt from ERG Guide 122 [Gases - Oxidizing (Including Refrigerated Liquids)]:· Substance does not burn but will support combustion.Not combustible but enhances combustion of other substances. Heating will cause rise in pressure with risk of bursting.Not combustible but enhances combustion of other substances. Risk of fire and explosion on contact with combustible substances or reducing agents. |
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| Brandpotenzial | Gas: moderate fire risk as oxidizing agent.Oxygen is noncombustible, but actively supports combustion.Strong oxidizer ... May initiate fire/explosion in combustible materials. /Oxygen, liquid/Even a slight increase in the oxygen content of air above its usual 21 vol % will greatly increase the rate of oxidation or combustion of many substances.... Fires at the interface of oxygen regulators and cylinder valves because of incorrect use of /the plastic (usually Nylon) crush gasket of/ CGA 870 seals /was alerted/ ... 12 reports /had been received/ in which regulators used with oxygen cylinders have burned or exploded, in some cases injuring personnel. Some of the incidents occurred during emergency medical use or during routine equipment checks.... 16 reports of aluminum regulators used with oxygen cylinders burning or exploding /had been received/. These incidents caused severe burns to 11 health care workers and patients. Many of the incidents occurred during emergency medical use or during routine equipment checkout. FDA and The National Institute for Occupational Safety and Health (NIOSH) believe that the aluminum in these regulators was a major factor in both the ignition and severity of the fires ... |
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| Gesundheitsgefahren | Inhalation of 100% oxygen can cause nausea, dizziness, irritation of lungs, pulmonary edema, pneumonia, and collapse. Liquid may cause frostbite of eyes and skin. (USCG, 1999)Excerpt from ERG Guide 122 [Gases - Oxidizing (Including Refrigerated Liquids)]:· Vapors may cause dizziness or asphyxiation without warning, especially when in closed or confined areas. |
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| Gefahrenübersicht | A powerful oxidizer and explosion hazardLiquid may cause frostbiteInhalation of 100% oxygen for 24 hours can cause pulmonary edemaChronic exposure to high concentrations of oxygen can cause lung scarring[CHEMINFO] Oxygen toxicity is observed in mechanically ventilated patients treated for hypoxic respiratory failure. Exposures to oxygen at FIO2 of 50 to 60% for more than 6 hours can cause lung injury including alveolar edema, formation of hyaline membranes, hypoxemia, and progressive fibrosis. [Cecil, p. 582] |
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| DOT-Nummer und Handbuch | 1072 1221073 122 |
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| Reaktive Gruppe | Oxidizing Agents, Strong |
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| EG-Einstufung | Symbol: OR: 8S: (2)-17 |
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| UN-Einstufung | UN Hazard Class: 2.2UN Subsidiary Risks: 5.1 |
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| Special Reports | A REVIEW WITH 123 REFERENCES ON THE MECHANISMS OF OXYGEN TOXICITY & PROPOSED MEANS OF PROTECTION AGAINST ITS DETRIMENTAL ACTIONS.[FRANK L, MASSARO DAM J MED 69 (1): 117-26 (1980)] |
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| Brandbekämpfung | Excerpt from ERG Guide 122 [Gases - Oxidizing (Including Refrigerated Liquids)]:In case of fire in the surroundings, use appropriate extinguishing media. In case of fire: keep cylinder cool by spraying with water. Combat fire from a sheltered position.In case of fire in the surroundings, use appropriate extinguishing media. In case of fire: keep drums, etc., cool by spraying with water. NO direct contact with water. Combat fire from a sheltered position. |
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| Erste-Hilfe-Maßnahmen | Refer for medical attention.Fresh air, rest. Refer for medical attention.ON FROSTBITE: rinse with plenty of water, do NOT remove clothes. Refer for medical attention .First rinse with plenty of water for several minutes (remove contact lenses if easily possible), then refer for medical attention. |
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| Freisetungsmaßnahmen | · CALL 911. Then call emergency response telephone number on shipping paper. If shipping paper not available or no answer, refer to appropriate telephone number listed on the inside back cover.· Keep combustibles (wood, paper, oil, etc.) away from spilled material. |
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| Erste Hilfe | INHALATION: in all but the most severe cases (pneumonia), recovery is rapid after reduction of oxygen pressuresupportive treatment should include immediate sedation, anticonvulsive therapy if needed, and rest.Excerpt from ERG Guide 122 [Gases - Oxidizing (Including Refrigerated Liquids)]:General First Aid: |
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| Sichere Lagerung | Fireproof. Separated from combustible substances and reducing agents. Cool. |
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| Firefighting Hazards | If oxygen-enriched clothing catches fire, extinguish under safety showerfire blanket may not be effective. Use continuous water spray to soak clothing of a rescuer who must operate in oxygen-enriched fire area.Most fibrous fabrics will absorb oxygen when exposed to concn greater than the normal 21% in air, and this is retained for a long time after excess oxygen is no longer present, greatly increasing the possibility of ignition.Heating will cause rise in pressure with risk of bursting ... The gas is heavier than air. |
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| Expositionsschutz und Persönliche Schutzausrüstung | · Wear positive pressure self-contained breathing apparatus (SCBA). |
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| Brandbekämpfungsverfahren | LIQ: When fire results from a leak or flow of liq oxygen onto wood, paper, waste or another similar material, the first thing to do is stop flow if possible. For small spills, or after leak or flow of liq oxygen has been stopped, use enough water to put out fire quickly. When fire involves liq oxygen and liq fuels, control it as follows: (a) When liq oxygen leaks or flows into large quantities of fuel, shut off flow of liq oxygen, and put remaining fuel fire out with extinguishing agents suitable for use on class B fires. When fuel leaks or flows into large quantities of liq oxygen, shut off flow of fuel. (b) When fuel and liq oxygen are mixed or mixing but are not yet burning, isolate area from sources of ignition and get out quickly, allowing oxygen to evaporate. When large pools of water-soluble fuel are present, use water to dilute fuel and reduce intensity of fire. This method cannot be used with fuels which do not mix with water. Appropriate extinguishing agents may be used to put out fuel fires after the oxygen has evaporated.If material involved in fire: Dangerously explosive. Cool all affected containers with flooding quantities or water. Do not use water on material itself. Apply water from as far a distance as possible. /Oxygen, refrigerated liquid/If material involved in fire: Dangerously explosive. Cool all affected containers with flooding quantities of water. Apply water from as far as distance as possible. /Oxygen, compressed/Evacuation: If fire becomes uncontrollable or container is exposed to direct flame: consider evacuation of one-third (1/3) mile radius. /Oxygen, compressedOxygen, refrigerated liquid/ |
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| Lagerbedingungen | GAS & LIQ: GASEOUS OXYGEN IS STORED ... IN CYLINDERS AT A PRESSURE OF 150-160 ATM, & INSULATED TANKS ARE USED FOR LIQUID OXYGENSMALL QUANTITIES OF LIQUID OXYGEN (2-50 L) CAN BE STORED IN DEWAR FLASKS.Oxygen should be stored in an area that is at least 20 ft away from any flammable or combustible materials (especially oil & grease) or separated from them by a noncombustible barrier at least 5 ft high & having a fire-resistant rating of at least 1/2 hr.LIQ: Protect against physical damage. Isolate from combustible gas installations and combustible materials by adequate distance or by gas-tight fire-resistive barriers. Protect against overheating. Outside storage of liq oxygen tanks is recommended.Where oxygen may be released, provide adequate ventilation to prevent excessive oxygen-enrichment of the workplace atmosphere (holding at < 23 Vol % O2 is recommended for fire safety).Store oxygen containers in a clean, cool, dry, well-ventilated, low-fire risk area ... Never expose any part of a cylinder to temperature above 125 °F. Ground equipment to eliminate build-up of static charge. Ensure that containers of liquid oxygen are properly vented to prevent pressure build-up and that suitable materials are used to contact liquid oxygen and high purity oxygen. /Liquid and compressed oxygen/ |
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| Reinigungsverfahren | Notify safety personnel of significant leaks or spills. ... Shut off oxygen source if possible.To increase the rate of controlled evaporation, spray with large amounts of water (fog may be generated and reduce visibility). /Liquid oxygen/Eliminate all ignition sources. Stop or control the leak, if this can be done without undue risk. USe water spray to disperse vapors and protect personnel.Ventilation. Remove all ignition sources. Do NOT absorb in saw-dust or other combustible absorbents. NEVER direct water jet on liquid. /Oxygen, liquefied/ |
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| Freisetung ohne Brand | Excerpt from ERG Guide 122 [Gases - Oxidizing (Including Refrigerated Liquids)]: |
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| Entsorgungsverfahren | SRP: The most favorable course of action is to use an alternative chemical product with less inherent propensity for occupational exposure or environmental contamination. Recycle any unused portion of the material for its approved use or return it to the manufacturer or supplier. Ultimate disposal of the chemical must consider: the material's impact on air qualitypotential migration in soil or watereffects on animal, aquatic, and plant lifeand conformance with environmental and public health regulations.Remove waste containers or leaking cylinders to exhaust hood or outdoors away from combustibles and allow to discharge at moderate rate. Tag cylinder to indicate defect, close valve and return to supplier. /Liquid and compressed oxygen/Evaporation: Remove waste containers or leaking cylinders to exhaust hoods or outdoors away from combustibles and allow to discharge @ a moderate rate. Tag cylinder to indicate defect, close valve and return to supplier. |
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| Wechselwirkungen | ... Toxicity of oxygen to dogs' eyes could be enhanced ... by promazine ... chlorpromazine, thioridazine, & chloroquine.... Some anticancer drugs, for example, bleomycin and cyclophosphamide, show increased acute lung damage in animals in combination with hyperoxia. Whether this is also true in humans is uncertain. It appears that direct interaction between O2 and the anticancer drugs occurs, rather than interference by O2 with the repair of damage done by the drugs.Hyperoxia and nitric oxide act synergistically in their cytotoxicity to A549 cells (human alveolar epithelial cells). This interaction is of human relevance because nitric oxide is being tested clinically for infants and children with pulmonary hypertension, and these patients are also frequently receiving simultaneous O2 therapy.... Carefully maintained, anesthetized, intubated, and mechanically ventilated baboons /were exposed/ for 11 days to 40% O2, 80% O2, 100% O2 ... or 80% O2 followed by inoculation with Pseudomonas aeruginosa bacteria. The groups exposed to 100% O2 or 80% O2 plus bacterial inoculation showed mixed exudative-reparative diffuse alveolar lesions, altered morphology of type II cells, increased numbers of type II cells and interstitial cells, and decreased numbers of type I and endothelial cells. The animals exposed to 40% O2 or 80% O2 showed increased numbers of alveolar macrophages and focal widening of alveolar walls. The most striking finding was that 80% O2 plus infection caused responses as severe as 100% O2. It was suggested that the combination with infection reflects the evolution of adult respiratory distress syndrome that occurs in some human patients in intensive medical care units (and other situations involving lung injury).For more Interactions (Complete) data for OXYGEN (25 total), please visit the HSDB record page. |
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| Expositionspfade | The substance can be absorbed into the body by inhalation. |
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| Signs and Symptoms | Cough. Dizziness. Sore throat. Visual disturbances.Cough. Dizziness. Sore throat.ON CONTACT WITH LIQUID: FROSTBITE.See Skin. |
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| Natural Pollution Sources | ... oxygen, as a gaseous element, forms 21% of atmosphere by volume.EVEN THOUGH LARGE QUANTITIES OF ATMOSPHERIC O2 ARE CONSTANTLY BEING CONSUMED IN RESPIRATION, COMBUSTION & OTHER OXIDATION PROCESSES, THE CONCN OF O2 IS KEPT @ VIRTUALLY CONSTANT LEVEL, PRIMARILY AS A RESULT OF O2 LIBERATED IN THE PROCESS OF PHOTOSYNTHESIS IN GREEN PLANTS. |
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| Atmospheric Concentrations | Normal air consists of 20.94% oxygen. |
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| Human Toxicity Excerpts | /HUMAN EXPOSURE STUDIES/ "Hyperbaric" is an atmospheric pressure greater than that at sea level. This increase in pressure, often associated with diving, is opposite of the decrease in atmospheric pressure associated with aviation and mountain climbing (hypobaric). Partial pressures of O2 are higher in hyperbaric, and lower in hypobaric, atmospheres. Increased O2 pressures result in hyperoxygenation of the blood (increased dissolved O2) and allows for improved O2 delivery to tissues. Hyperbaric O2 has a number of important uses in medicine. However, the toxicity of increased O2 pressures are also evident in human and animal studies./HUMAN EXPOSURE STUDIES/ As part of a comprehensive study of specific organ oxygen tolerance, oxygen effects on pulmonary function were measured in normal, resting men who breathed oxygen continuously at 3.0, 2.5, 2.0, and 1.5 atmospheres absolute for average durations of 3.4, 5.7, 9.0, and 17.7 hr, respectively. Rates of development of effects of pulmonary oxygen toxicity were monitored during oxygen exposure at 2.5, 2.0, and 1.5 atmospheres absolute with repeated flow-volume loops, spirometry, and symptom assessment. Additional pulmonary measurements before and after exposure included lung compliance, airway resistance, density dependence of flow, nitrogen closing volumes, carbon monoxide diffusing capacity, and alveolar-arterial oxygen differences. Of these measurements only airway resistance and closing volumes were not significantly affected at any pressure, but patterns and magnitudes of effects varied at different pressures. Overall, the data indicate that continuous oxygen exposure at 3.0 to 1.5 atmospheres absolute affects pulmonary mechanical function earlier and more prominently than carbon monoxide diffusing capacity. Recovery of lung mechanical function usually occurred within 12-24 hr after exposure, but required more than 24 hr in some individuals. No individual measure of pulmonary function was found to be uniquely satisfactory for monitoring rates of development or reversal of pulmonary oxygen poisoning. The existence of multiple pulmonary effects of oxygen toxicity and the complexity of their interactions require selective applications of individual toxicity indices to specific conditions of exposure and recovery./HUMAN EXPOSURE STUDIES/ ... Direct visual observations /were made/ of the interior of the trachea and ... rate of mucus transport /measured/ in 10 human volunteers who breathed oxygen through a face mask for 6 hr. The oxygen concn was 90 to 95%. Observations were made after 3 and 6 hr. Tracheal mucous velocity was decreased after 3 hr and all subjects showed visual evidence of tracheal irritation after 6 hr. ... Three subjects had fever and two had symptoms of acute bronchitis several hours after the study was completedone had sinusitis and conjunctivitis. These phenomena might be related to the procedure of passing a fiberoptic bronchoscope into the trachea. Two others became nauseated and vomited during the evening of the study. All were extremely fatigued. None complained of substernal pain or other symptoms of tracheal or bronchial irritation during the oxygen exposure./HUMAN EXPOSURE STUDIES/ To study the early changes in the lower respiratory tract in persons exposed to periods of hyperoxia usually considered safe, 14 normal subjects were evaluated by bronchoalveolar lavage before and immediately after 16.7 +/ - 1.1 hours of breathing more than 95% oxygen. Hyperoxia caused a significant alveolar-capillary "leak" as detected by the presence of increased plasma albumin and transferrin in lavage fluid. Although some of the effects of exposure to 17 hr of more than 95% oxygen are reversible, hyperoxia for even this short period lowers the structural or functional barriers that normally prevent alveolar-capillary "leak" and induces processes that can culminate in fibrosis of the alveolar wall.For more Human Toxicity Excerpts (Complete) data for OXYGEN (33 total), please visit the HSDB record page. |
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| Environmental Bioconcentration | None /Liquid oxygen/ |
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| Non-Human Toxicity Excerpts | /LABORATORY ANIMALS: Acute Exposure/ ... One-day-old rats were exposed to 2 to 4 days of /normobaric hyperoxia/ (NH) (Fio2>0.98) or normoxia (Fio2=0.21), with or without weaning. Pups were then euthanized and 100 uL of blood was collected (cardiac puncture) for differential white blood cells analysis (n=12 per group). The lungs, a piece of distal ileum, and the left kidney were removed for histologic evaluation. Both NH and weaning /(return to air breathing)/ generated significant increases in blood neutrophil count, whereas lymphocyte population was significantly increased only after weaning (P<0.05analysis of variance with Bonferroni correction for multiple comparisons). Normobaric hyperoxia created mild increases in the renal tubular necrosis, dilation, regeneration, and interstitial inflammation. A significant increase in the intestinal serosal and submucosal vasodialation and vascularization occurred 1 day after weaning from 4 days of NH (P<0.001)./LABORATORY ANIMALS: Acute Exposure/ /Dogs inhaling pure O2 at atmospheric pressure/ ... poisoning begins after 36 hr, causes distress within 48 hr, and death in 60 hr. Ninety per cent O2 in air requires double exposure period for similar resultsin 80% oxygen in air the animals did not die but were ill at the end of a continuous exposure of 1 wk. A decline in O2 saturation of blood, rise in hemoglobin, lung congestion and edema, right-heart failure, and liver congestion were frequent findings in oxygen poisoning./LABORATORY ANIMALS: Acute Exposure/ The role of neutrophils in the mediation of severe normobaric hyperoxic lung injury was studied by monitoring the effects of neutrophil depletion on a rat model of pulmonary oxygen toxicity. Pulmonary capillary permeability, assessed using an radioactive iodine labeled albumin lung permeability index, progressively increased with an increased duration of hyperoxic exposure in normal animals (lung permeability index of 0.43 +/ - 0.09 at 2 hr0.95 +/ - 0.17 at 48 hr1.56 +/ - 0.21 at 60 hr) despite the absence of any significant tissue or bronchoalveolar lavage evidence of neutrophil infiltration until 60 hr of hyperoxia exposure. Neutrophil depletion with cyclophosphamide blocked this late neutrophil infiltrate but failed to attenuate lung injury./LABORATORY ANIMALS: Acute Exposure/ One litter of term newborn New Zealand albino rabbits (n= 9) and 15 adult rabbits were exposed to 65 hr of > 95% oxygen (O2). ... Sixty-five hr of oxygen exposure in newborn rabbits produced no evidence of lung injury on light microscopy, 97% of bronchoalveolar lavage white cells were alveolar macrophages and bronchoalveolar lavage protein was low. No differences were found in the percentage of alveolar macrophages, and the proportion of PMN was low in both hyperoxic (n=9) and normoxic (n=7) newborn groups. Mortality was comparable, 14% and 11% in normoxic and hyperoxic newborn groups, respectively. An equal period of oxygen exposure produced significant lung injury in adult rabbits, and a mortality of 27% (not significant). Adults appeared dyspneic and frequently had grossly visible subpleural hemorrhages. Microscopic analysis revealed an intense inflammatory cell infiltrate, an exudate in the air spaces and areas of intra-alveolar hemorrhage. Bronchoalveolar lavage fluid from oxygen injured adults contained a 17-fold greater percentage of PMN and 16-fold higher protein than oxygen exposed newborns. Newborns under normoxic conditions had very high levels of 6-keto-PGF1a and low quantities of TXB2. Hyperoxic newborns had lower 6-keto-PGF1a and significantly higher TXB2. Hyperoxic adults had significantly lower 6-keto-PGF1a, and significantly higher LTB4 and LTC4 in bronchoalveolar lavage compared to hyperoxic newborns. Leukotriene D4 values, although 25% higher on average in the adult rabbits, were not statistically different.For more Non-Human Toxicity Excerpts (Complete) data for OXYGEN (29 total), please visit the HSDB record page. |
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| Populations at Special Risk | Ventilating patients with elevated oxygen tensions alters normal respiratory physiology and may damage lung tissue, depending on coexisting host and iatrogenic factors. ... Supplemental oxygen should be prescribed at the lowest concentration possible that will still allow adequate tissue oxygenation...In a small number of patients whose respiratory center is depressed by long-term retention of carbon dioxide, injury, or drugs, ventilation is maintained largely by stimulation of carotid and aortic chemoreceptors, commonly referred to as the hypoxic drive. The provision of too much oxygen can depress this drive, resulting in respiratory acidosis. In these cases, supplemental oxygen should be titrated carefully to ensure adequate arterial saturation. If hypoventilation results, then mechanical ventilatory support with or without tracheal intubation should be provided.... Premature infants have organs that are not fully developed at birth, including the lungs and eyes ... so they are placed in enclosed incubators or on respirators with an increased PO2. In the early history of this life-saving procedure, high O2 concentrations were used. The immediate benefit of tissue oxygenation was followed by a high incidence of pulmonary damage. It was soon observed that these babies were blind, the result of hyperoxic damage to the retinal capillaries. This injury was followed by proliferation of fibrous tissue. Careful adjustment of the O2 pressure to meet the needs of the infant has been effective in controlling the damage to the eye.... Premature infants ... placed in incubators ... breathe oxygen in concentrations greater than in air. On removal from hyperoxia, they develop irreversible bilateral ocular disease known as retrolental fibroplasia ... . Only the incompletely developed retinal circulation is susceptible to toxic levels of oxygen, whereas a mature retinal vascular system and other incompletely formed circulations are not sensitive to oxygen toxicity. Within 6 hr after an infant is placed in a high oxygen containing atmosphere, vasoconstriction of the immature vessels occurs, which is reversible if the child is immediately returned to air but is irreversible if hyperoxia therapy is continued.For more Populations at Special Risk (Complete) data for OXYGEN (7 total), please visit the HSDB record page. |
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| Probable Routes of Human Exposure | Industrial exposures to high oxygen pressure are uncommon. Sea diving is probably the most frequent. ... Caison workers & tunnel makers may also be exposed to pressures that are high enough to cause lung damage.Some potential risks for intoxication with oxygen also exist for drivers and persons living or working in closed compartments, where the air is reconditioned by the addition of pure oxygen (eg, submarines and spacecraft), should the regulation system malfunction. |
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| Antidote and Emergency Treatment | Some protection against oxygen toxicity syndrome is offered by admin of gamma-aminobutyric acid, succinate, chelating agents, certain anesthetics, and trimethamine (tris-(hydroxymethyl)aminomethane).Contact with liquid oxygen: Remove victim from source of contact. Flush affected areas with lots of tepid water (Do not apply directly to affected area). Loosely apply dry sterile, bulky dressings to protect area from infection/injury. Get medical attention. /Liquid oxygen/Basic treatment: Establish a patent airway (oropharyngeal or nasopharyngeal airway, if needed). Suction if necessary. Watch for signs of respiratory insufficiency and assist ventilations if necessary. Administer oxygen by nonrebreather mask at 10 to 15 L/min. Monitor for pulmonary edema and treat if necessary ... . Monitor for shock and treat if necessary ... . For eye contamination, flush eyes immediately with water. Irrigate each eye continuously with 0.9% saline (NS) during transport ... . Do not use emetics. For ingestion, rinse mouth and administer 5 ml/kg up to 200 ml of water for dilution if the patient can swallow, has a strong gag reflex, and does not drool ... . Do not attempt to neutralize because of exothermic reaction. Cover skin burns with dry, sterile dressings after decontamination ... . /Oxidizers/Advanced treatment: Consider orotracheal or nasotracheal intubation for airway control in the patient who is unconscious, has severe pulmonary edema, or is in severe respiratory distress. Early intubation, at the first sign of upper airway obstruction, may be necessary. Positive-pressure ventilation techniques with a bag valve mask device may be beneficial. Consider drug therapy for pulmonary edema ... . Monitor cardiac rhythm and treat arrhythmias as necessary ... . Start IV administration of D5W /SRP: "To keep open", minimal flow rate/. Use 0.9% saline (NS) or lactated Ringer's if signs of hypovolemia are present. For hypotension with signs of hypovolemia, administer fluid cautiously. Watch for signs of fluid overload ... . Use proparacaine hydrochloride to assist eye irrigation ... . /Oxidizers/ON FROSTBITE: rinse with plenty of water, do NOT remove clothes. Refer for medical attention ... Eye exposure: First rinse with plenty of water for several minutes (remove contact lenses if easily possible), then take to a doctor. /Oxygen, liquefied/ |
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| UN-Transportnummer | UN 1073 |
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| Gefahrenklasse | 2.2 |
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