1306-24-7[Se]=[Cd]AQCDIIAORKRFCD-UHFFFAOYSA-NCadmium selenide is a cadmium molecular entity.
1306-24-7、157453-13-9、331269-19-3DTXSID1061651CHEBI:50834Dry Powder
White to brown solid; Turns red in sunlight; [Merck Index] Red or black odorless powder; Insoluble in water; [MSDSonline]
안전 정보는 참고용이며, 공급업체의 물질안전보건자료(SDS), 관련 법규 및 전문가 평가를 대체하지 않습니다.
Danger
H301+H331 (32.5%): Toxic if swallowed or if inhaled [Danger Acute toxicity, oral; acute toxicity, inhalation]
P203, P260, P261, P264, P270, P271, P273, P280, P301+P316, P302+P352, P304+P340, P316, P317, P318, P319, P321, P330, P362+P364, P391, P403+P233, P405, and P501 (click each P-code to see the statement)
Aggregated GHS information provided per 323 reports by companies from 12 notifications to the ECHA C&L Inventory. Each notification may be associated with multiple companies.
Danger
H302: Harmful if swallowed [Warning Acute toxicity, oral]
Chemical: Cadmium selenide (CdSe)
Cadmium selenide (CdSe) is listed on the EPA's Chemical Data Reporting (CDR) system. Manufacturers and importers of Cadmium selenide (CdSe) are required to report information about their production and use of this chemical to the EPA under the Toxic Substances Control Act (TSCA). (40 eCFR Part 711)
Status: Active Update: 04-10-2022 https://echa.europa.eu/registration-dossier/-/registered-dossier/23754
Restricted substance: Cadmium selenide
Cadmium selenide: Does not have an individual approval but may be used under an appropriate group standard
Acute Tox. 3 (98.1%)
Acute toxicity - category 4
Cadmium is absorbed from oral, inhalation, and dermal routes. Cadmium initially binds to metallothionein and albumin and is transported mainly to the kidney and liver. Toxic effects are observed once the concentration of cadmium exceeds that of available metallothionein, and it has also been shown that the cadmium-metallothionein complex may be damaging. Cadmium is not known to undergo any direct metabolic conversion and is excreted unchanged, mainly in the urine. Selenium may be absorbed through inhalation and ingestion, while some selenium compounds may also be absorbed dermally. Once in the body, selenium is distributed mainly to the liver and kidney. Selenium is an essential micronutrient and is a component of glutathione peroxidase, iodothyronine 5'-deiodinases, and thioredoxin reductase. Organic selenium is first metabolized into inorganic selenium. Inorganic selenium is reduced stepwise to the intermediate hydrogen selenide, which is either incorporated into selenoproteins after being transformed to selenophosphate and selenocysteinyl tRNA or excreted into the urine after being transformed into methylated metabolites of selenide. Elemental selenium is also methylated before excretion. Selenium is primarily eliminated in the urine and feces, but certain selenium compounds may also be exhaled. (L619, L6)
Used in photoconductors, semiconductors, photoelectric cells, rectifiers, and phosphors; [Merck Index] Used as red pigment; [Hawley]
Semiconductor Manufacturing [Category: Industry]
Cadmium selenide is used as a semiconductor. It is also found in laser diodes. (L471)
현재 필터 조건에 해당하는 반응 기록이 없습니다.
불러오는 중…
3–12mm pieces (sintered), and -325 mesh 10μm or less with 99.999% purity; white to brown; cub or hexagonal crystal(s); hex: a=0.4309nm, c=0.7021 nm, enthalpy of fusion 305.307 kJ/mol; becomes red in sunlight; used as a red pigment, in semiconductors, and as an evaporation material and sputtering target to produce photoconductive films and infrared filters; possible use in the electrofabrication of microdiode arrays [HAW93] [MER06] [CER91] [KLE93] [KRE91]
Biological Exposure Indices (BEI) [ACGIH] - Cd in urine = 5 ug/g creatinine; Cd in blood = 5 ug/L; sampling time not critical; Monitoring in blood should be preferred during the initial year of exposure and whenever changes in the degree of exposure are suspected. [ACGIH]
0.02 [mg/m3], inhalable fraction, as Se[German Research Foundation (DFG)]
Acute Inhalation: 0.00003 mg/m3 (Cadmium) (L134) Chronic Inhalation: 0.00001 mg/m3 (Cadmium) (L134) Intermediate Oral: 0.0005 mg/kg/day (Cadmium) (L134) Chronic Oral: 0.0001 mg/kg/day (Cadmium) (L134) Chronic Oral: 0.005 mg/kg/day (Selenium) (L134)
Chronic exposure to cadmium fumes can cause chemical pneumonitis, pulmonary edema, and lung diseases such as bronchitis and emphysema. Cadmium also accumulates in the kidneys, causing permanent damage. Loss of bone density also occurs. Chronic oral exposure to high concentrations of selenium compounds can produce a disease called selenosis. The major signs of selenosis are hair loss, nail brittleness, and neurological abnormalities (such as numbness and other odd sensations in the extremities). Animal studies have shown that selenium may also affect sperm production and the female reproductive cycle. (L619, L6)
Cadmium initially binds to metallothionein and is transported to the kidney. Toxic effects are observed once the concentration of cadmium exceeds that of available metallothionein, and it has also been shown that the cadmium-metallothionein complex may be damaging. Accumulation of cadmium in the kidney results in increased excretion of vital low and high weight molecular proteins. Cadmium is a high affinity zinc analog and can interfere in its biological processes. It also binds to and activates the estrogen receptor, likely stimulating the growth of certain types of cancer cells and causing other estrogenic effects, such as reproductive dysfunction. Cadmium causes cell apoptosis by activating mitogen-activated protein kinases. Selenium readily substitutes for sulfur in biomolecules and in many biochemical reactions, especially when the concentration of selenium is high and the concentration of sulfur is low. Inactivation of the sulfhydryl enzymes necessary for oxidative reactions in cellular respiration, through effects on mitochondrial and microsomal electron transport, might contribute to acute selenium toxicity. Selenomethionine (a common organic selenium compound) also appears to randomly substitute for methionine in protein synthesis. This substitution may affect the structure and functionability of the protein, for example, by altering disulfide bridges. Inorganic forms of selenium appear to react with tissue thiols by redox catalysis, resulting in formation of reactive oxygen species and causing damage by oxidative stress. (L619, L8, A18, A19, A28)
Acute inhalation of cadmium fumes results in metal fume fever, which is characterized by chills, fever, headache, weakness, dryness of the nose and throat, chest pain, and coughing. Ingestion of cadmium causes vomiting and diarrhea. Short-term oral exposure to high concentrations of selenium may cause nausea, vomiting, and diarrhea. Brief exposures to high levels of elemental selenium or selenium dioxide in air can result in respiratory tract irritation, bronchitis, difficulty breathing, and stomach pains. Longer-term exposure to either of these air-borne forms can cause respiratory irritation, bronchial spasms, and coughing. (L619, L6)
Electrical Equipment, Appliance, and Component Manufacturing
Cadmium selenide (CdSe): ACTIVE
개요Cadmium selenide (CdSe) appears as grayish-brown or red crystals, is insoluble in water, highly toxic, and has a melting point of 1350°C. It can be produced by co-heating metallic cadmium with hydrogen selenide and is used in electron emitters, spectral analysis, photoconductors, semiconductors, and photosensitive components.
용도Cadmium selenide is used as a n-type semiconductor and its nanoparticles are used in laser diodes, opto-electronic devices, nanosensing, biomedical imaging and high efficiency solar cells. Since, it is transparent to infrared light, it finds application in photoresistors and windows for instruments utilizing IR light.
일반 설명CdSe crystal can exist in solid hexagonal or cubic structure. CdSe behaves as an n-type semiconductor, with a bandgap of 1.74 eV (300K).1