납(II)NTA.

SRP: Contaminated protective clothing should be segregated in a manner so that results in no direct personal contact by personnel who handle, dispose of, or clean the clothing. Quality assurance procedures to confirm the efficacy of the cleaning procedures should be implemented prior to the decontaminated protective clothing being returned for reuse by the workers. Contaminated clothing (including shoes/socks) should not be taken home at end of shift, but should remain at employee's place of work for cleaning.

SRP: Wastewater from contaminant suppression, cleaning of protective clothing/equipment, or contaminated sites should be contained and evaluated for subject chemical or decomposition product concentrations. Concentrations shall be lower than applicable environmental discharge or disposal criteria. Alternatively, pretreatment and/or discharge to a permitted wastewater treatment facility is acceptable only after review by the governing authority and assurance that "pass through" violations will not occur. Due consideration shall be given to remediation worker exposure (inhalation, dermal and ingestion) as well as fate during treatment, transfer and disposal. If it is not practicable to manage the chemical in this fashion, it must be evaluated in accordance with EPA 40 CFR Part 261, specifically Subpart B, in order to determine the appropriate local, state and federal requirements for disposal.

The worker should wash daily at the end of each work shift, and prior to eating, drinking, smoking, etc. /Lead/

Work clothing that becomes wet or significantly contaminated should be removed or replaced. /Lead/

Workers whose clothing may have become contaminated should change into uncontaminated clothing before leaving the work premises. /Lead/

0.05 [mg/m3], as Pb

8 hr Time Weighted Avg (TWA): 0.05 mg/cu m. /Lead, and inorganic compounds, as Pb/

Excursion Limit Recommendation: Excursions in worker exposure levels may exceed three times the TLV-TWA for no more than a total of 30 min during a work day, and under no circumstances should they exceed five times the TLV-TWA, provided that the TLV-TWA is not exceeded. /Lead, inorganic compounds, as Pb/

A3; Confirmed animal carcinogen with unknown relevance to humans.

Biological Exposure Index (BEI): Determinant: lead in blood; Sampling Time: not critical; BEI: 30 ug/100 mL. [Note: Women of child bearing potential, whose blood Pb exceeds 10 ug/dL, are at risk of delivering a child with a blood Pb over the current Centers for Disease Control guideline of 10 ug/dL. If the blood Pb of such children remains elevated, they may be at increased risk of cognitive deficits. The blood Pb of these children should be closely monitored and appropriate steps should be taken to minimize the child's exposure to environmental lead.] /Lead, and inorganic compounds, as Pb/

Wear appropriate personal protective clothing to prevent skin contact. /Lead/

Wear appropriate eye protection to prevent eye contact. /Lead/

Respirator Recommendations: Up to 0.5 mg/cu m. /Lead/ [Table#3081]

Respirator Recommendations: Up to 1.25 mg/cu m. /Lead/ [Table#3082]

For more Personal Protective Equipment (PPE) (Complete) data for LEAD (2+) NTA (9 total), please visit the HSDB record page.

Residual lead levels. The /U.S. Fish and Wildlife Service's/ maximum environmentally acceptable level of lead in shot is trace amounts or <1 percent. Any shot manufactured with lead levels equal to or exceeding 1 percent are considered toxic and, therefore, illegal.

In this part 1303, the Consumer Product Safety Commission declares that paint and similar surface-coating materials for consumer use that contain lead or lead compounds and in which the lead content (calculated as lead metal) is in excess of 0.06 percent (0.06 percent is reduced to 0.009 percent effective August 14, 2009 as mandated by Congress in section 101(f) of the Consumer Product Safety Improvement Act of 2008, Pub. L. 110-314) of the weight of the total nonvolatile content of the paint or the weight of the dried paint film (which paint and similar surface-coating materials are referred to hereafter as "lead-containing paint") are banned hazardous products under sections 8 and 9 of the Consumer Product Safety Act (CPSA), 15 U.S.C. 2057, 2058. The following consumer products are also declared to be banned hazardous products: (1) Toys and other articles intended for use by children that bear "lead-containing paint". (2) Furniture articles for consumer use that bear "lead-containing paint".

Disclosure of Known Lead-Based Paint and/or Lead-Based Paint Hazards Upon Sale or Lease of Residential Property: This subpart implements the provisions of 42 U.S.C. 4852d, which impose certain requirements on the sale or lease of target housing. Under this subpart, a seller or lessor of target housing shall disclose to the purchaser or lessee the presence of any known lead-based paint and/or lead-based paint hazards; provide available records and reports; provide the purchaser or lessee with a lead hazard information pamphlet; give purchasers a 10-day opportunity to conduct a risk assessment or inspection; and attach specific disclosure and warning language to the sales or leasing contract before the purchaser or lessee is obligated under a contract to purchase or lease target housing.

Establishes standards for lead-based paint hazards in most pre-1978 housing and child-occupied facilities. This regulation supports the implementation of regulations already promulgated, and others under development, which deal with worker training and certification, lead hazard disclosure in real estate transactions, requirements for lead cleanup under State authorities, lead hazard evaluation and control in Federally-owned housing prior to sale and housing receiving Federal assistance, and U.S. Department of Housing and Urban Development (HUD) grants to local jurisdictions to perform lead hazard control. Establishes, under authority of TSCA section 402, residential lead dust cleanup levels and amendments to dust and soil sampling requirements.

For more Other Standards Regulations and Guidelines (Complete) data for LEAD (2+) NTA (12 total), please visit the HSDB record page.

/AQUATIC SPECIES/ ... Carp (Cyprinus carpio) /were held/ for 48 hr in lead nitrate concn of between 0 and 20 mg lead/L, with and without one of the three complexans, EDTA, NTA, or DTPA. The accumulation of lead in both viscera and gills was dose-related, with the highest levels for viscera and gills being 86,000 and 4,560 mg/kg dry wt, respectively. The complexans reduced the uptake of lead at all dose levels. Concn in viscera ranged from 399 to 690 mg/kg for the three complexans, and 298 to 645 mg/kg in gills, after exposure to 20 mg/Lead (which had given the above levels without chelating agents). It is not clear whether the levels of lead in the gills represented uptake into the tissue or adsorption onto the exterior surfaces.

/AQUATIC SPECIES/ ... A natural phytoplankton community /was exposed/ to concn of an unspecified inorganic lead salt ranging from 5 x 10-7 to 5 x 10-4 M/L. The community of organisms, isolated from lake water, consisted mainly of diatoms: Tabellaria flocculosa (53% by vol), Synedra sp. (13%), and Asterionella formosa (7%). Other important constituent species were Cryptomonas spp., Rhodomonas minuta variety lacustris, Dinobryon divergens, small species of chryptomonades, Gymnodinium sp., and Mallomonas sp. The authors monitored photosynthetic activity as uptake of (14)C-labeled hydrogen carbonate over a 20 hr incubation. Photosynthetic carbon fixation was reduced in a dose-dependent manner throughout the exposure range of lead in the medium; the reduction was 90% relative to control cultures at the highest exposure of 5 x 10-4 M lead/L. The addition of lake sediment to treated cultures reduced the toxic effect of lead; addition to control cultures increased the photosynthetic carbon uptake by 19%. A similar reduction in the toxic effects of lead was seen on adding organic matter filtered out of the lake water and after adding the chelating agent nitrilotriacetic acid (NTA) at non- toxic levels. Neither of these two variables affected photosynthesis in control cultures. The NTA had the greatest effect on lead toxicity, virtually eliminating the effect of lead on photosynthesis.

/PLANTS/ Effects of lead (Pb) and chelators, such as EDTA, HEDTA, DTPA, NTA and citric acid, were studied to evaluate the growth potential of Sesbania drummondii in soils contaminated with high concentrations of Pb. S. drummondii seedlings were grown in soil containing 7.5 g Pb(NO(3))(2) and 0-10 mmol chelators/kg soil for a period of 2 and 4 weeks and assessed for growth profile (length of root and shoot), chlorophyll a fluorescence kinetics (F(v)/F(m) and F(v)/F(o)) and Pb accumulations in root and shoot. Growth of plants in the presence of Pb+chelators was significantly higher (P < 0.05) than the controls grown in the presence of Pb alone. F(v)/F(m) and F(v)/F(o) values of treated seedlings remained unaffected ... On application of chelators, while root uptake of Pb increased four-five folds, shoot accumulations increased up to 40-folds as compared to controls (Pb only) depending on the type of chelator used. Shoot accumulations of Pb varied from 0.1 to 0.42% (dry weight) depending on the concentration of chelators used.

/GENOTOXICITY/ The frequencies of sister chromatid exchanges (SCE) were significantly increased in cultured Chinese hamster cells by insoluble salts of Cr(VI) (PbCrO4) and Pb(II) (PbSO4). A further significant increase of this effect was produced when diluted suspensions of PbCrO4 (1-4 mg/L) and PbSO4 (10-40 mg/L) were preincubated with nitrilotriacetic acid (NTA) concentrations (0.25-1mg/L) such as those that are possibly found in highly contaminated environmental situations. NTA enhanced the induction of SCE by PbCrO4 and PbSO4 even in the presence, in the preincubation solution and in the medium used to treat the cells, of Na+ and K+ concentrations largely exceeding (e.g., 10+2 to 10+6 times) those of the genotoxic metals.

A3; Confirmed animal carcinogen with unknown relevance to humans.

Lead, lead compounds: Reasonably anticipated to be a human carcinogen

Immediate first aid: Ensure that adequate decontamination has been carried out. If patient is not breathing, start artificial respiration, preferably with a demand-valve resuscitator, bag-valve-mask device, or pocket mask, as trained. Perform CPR as necessary. Immediately flush contaminated eyes with gently flowing water. Do not induce vomiting. If vomiting occurs, lean patient forward or place on left side (head-down position, if possible) to maintain an open airway and prevent aspiration. Keep patient quiet and maintain normal body temperature. Obtain medical attention. /Lead and related compounds/

Basic treatment: Establish a patent airway (oropharyngeal or nasopharyngeal airway, if needed). Suction if necessary. Watch for signs of respiratory insufficiency and assist ventilations if necessary. Administer oxygen by nonrebreather mask at 10 to 15 L/min. Monitor for shock and treat if necessary ... . Anticipate seizures and treat if necessary ... . For eye contamination, flush eyes immediately with water. Irrigate each eye continuously with 0.9% saline (NS) during transport ... . Do not use emetics. For ingestion, rinse mouth and administer 5 ml/kg up to 200 ml of water for dilution if the patient can swallow, has a strong gag reflex, and does not drool ... . Administer activated charcoal ... . /Lead and related compounds/

Advanced treatment: Consider orotracheal or nasotracheal intubation for airway control in the patient who is unconscious or is in severe respiratory distress. Moderate hyperventilation (20 respiration per minute) may be beneficial for increased intracranial pressure. Start IV administration of 0.9% saline (NS) or lactated Ringer's (LR) /SRP: "To keep open", minimal flow rate/. For hypotension ... . Treat seizures with diazepam or lorazepam ... . Use proparacaine hydrochloride to assist eye irrigation ... . /Lead and related compounds/

Treatment. Emergency and supportive measures. Treat seizures and coma if they occur. Provide adequate fluids to maintain urine flow but avoid overhydration, which may aggravate cerebral edema. Avoid phenothiazines for delirium, as they may lower the seizure threshold. Patients with increased intracranial pressure may benefit from corticosteroids and mannitol. /Lead/

For more Antidote and Emergency Treatment (Complete) data for LEAD (2+) NTA (6 total), please visit the HSDB record page.

D008; A solid waste containing lead may or may not become characterized as a hazardous waste when subjected to the Toxicity Characteristic Leaching Procedure listed in 40 CFR 261.24, and if so characterized, must be managed as a hazardous waste. /Lead/

The Environmental Protection Agency is ... /promulgated/ regulations concerning ground-water monitoring with regard to screening suspected contamination at land based hazardous waste treatment, storage, and disposal facilities. ... /There are/ new requirements to analyze for a specified core list of chemicals plus those chemicals specified by the Regional Administrator on a site-specific basis. ... /Total lead (all species) is included on this list./ /Total lead (all species)/

National primary and secondary ambient air quality standards for lead and its compounds, measured as elemental lead by a reference method based on appendix G to this part, or by an equivalent method, are: 1.5 micrograms per cubic meter, maximum arithmetic mean averaged over a calendar quarter. /Lead and its compounds, as Pb/

Listed as a hazardous air pollutant (HAP) generally known or suspected to cause serious health problems. The Clean Air Act, as amended in 1990, directs EPA to set standards requiring major sources to sharply reduce routine emissions of toxic pollutants. EPA is required to establish and phase in specific performance based standards for all air emission sources that emit one or more of the listed pollutants. Lead compounds are included on this list. /Lead compounds/

(AZ) ARIZONA 20 ug/L /Lead/

(ME) MAINE 10 ug/L /Lead/